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  • Growing Evidence of a Proven Mechanism Shows Vitamin K2 Can . . .
    Arterial calcification is considered an unfortunate result of aging, but fortunately, we can actively regulate this process by providing our body with adequate amounts of vitamin K2 This nutrient is essential to activate the most potent modulator of vascular calcification–Matrix Gla Protein (MGP)–and thus lower the risk of age-related
  • Vitamin K2 and Arterial Calcification - Life Extension
    Vitamin K2 is required to activate proteins that defend against the deposition of calcium in blood vessels Vascular calcification is a major part of atherosclerosis, the number one contributor to heart attack and stroke Low vitamin K2 levels have been tied to higher risk for cardiovascular disease and higher rates of mortality
  • Vitamin K2 Reverses Artery Hardening by 40%
    Arterial calcification (AC) is generally regarded as an independent risk factor for cardiovascular morbidity and mortality Matrix Gla protein (MGP) is a potent inhibitor of AC, and its activity depends on vitamin K (VK) In rats, inactivation of MGP by treatment with the vitamin K antagonist warfarin leads to rapid calcification of the arteries
  • Can Vitamin K Help Protect Your Arteries from Calcification?
    The analysis found that vitamin K supplements—particularly K2—slowed the buildup of calcium in the arteries and improved levels of MGP, a protein that helps prevent calcification Importantly, the supplements were safe and well-tolerated
  • Power of Vitamin K2: Clearing Removing Calcium From Blood
    Osteocalcin: This protein, also activated by Vitamin K2, binds calcium to the bone matrix, promoting bone health and reducing arterial calcification The Pros of Vitamin K2 for Cardiovascular Health The benefits of Vitamin K2 for cardiovascular health are profound: Prevents Arterial Calcification: By activating MGP, Vitamin K2 helps prevent
  • Vitamin K2 and D in Patients With Aortic Valve Calcification . . .
    Background: Menaquinone-7 (MK-7), also known as vitamin K2, is a cofactor for the carboxylation of proteins involved in the inhibition of arterial calcification and has been suggested to reduce the progression rate of aortic valve calcification (AVC) in patients with aortic stenosis Methods: In a randomized, double-blind, multicenter trial, men from the community with an AVC score gt;300




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